Search results for "fetal programming"

showing 10 items of 13 documents

Genome-wide DNA methylation study in human placenta identifies novel loci associated with maternal smoking during pregnancy

2016

BACKGROUND: We conducted an epigenome-wide association study (EWAS) of DNA methylation in placenta in relation to maternal tobacco smoking during pregnancy and examined whether smoking-induced changes lead to low birthweight. METHODS: DNA methylation in placenta was measured using the Illumina HumanMethylation450 BeadChip in 179 participants from the INfancia y Medio Ambiente (INMA) birth cohort. Methylation levels across 431 311 CpGs were tested for differential methylation between smokers and non-smokers in pregnancy. We took forward three top-ranking loci for further validation and replication by bisulfite pyrosequencing using data of 248 additional participants of the INMA cohort. We ex…

0301 basic medicineAdultMaleplacentaEpidemiologyMaternal smokingPlacentaEpigenesis GeneticCohort Studies03 medical and health sciencesPregnancyTobacco SmokingMedicineBirth WeightHumansFetal programmingtobacco smokingCotinineDones embarassades -- Consum de tabacDNA methylationepigeneticsbusiness.industryInfant NewbornHuman placentaGeneral MedicineDNA MethylationMendelian Randomization Analysis030104 developmental biologyfetal programmingbirthweightMaternal ExposureSpainLinear ModelsChristian ministryCpG IslandsFemaleDNA -- MetilacióbusinessHumanitiesGenome-Wide Association Study
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The close link between the fetal programming imprinting and neurodegeneration in adulthood: The key role of “hemogenic endothelium” programming

2021

The research on neurodegenerative diseases (NeuroDegD) has been traditionally focused on later life stages. There is now an increasing evidence, that they may be programmed during early development. Here, we propose that NeuroDegD are the result of the complex process of imprinting on fetal hemogenic endothelium, from which the microglial cells make to origin. The central role of placenta and epigenetic mechanisms (methylation of DNA, histone modifications and regulation by non-coding RNAs) in mediating the short and long-term effects has been also described. Precisely, it reports their role in impacting plasticity and memory of microglial cells. In addition, we also underline the necessity…

0301 basic medicineAgingHemangioblastsCell PlasticityRisk AssessmentEpigenesis GeneticFetal DevelopmentMolecular Imprinting03 medical and health sciences0302 clinical medicineEpigenetic factors as biomarkers Sex dimorphism Fetal developmental programming Hemogenic endothelium Microglia plasticity and memory Neurodegenerative diseasesmedicineHumansSettore MED/05 - Patologia ClinicaEpigeneticsFetal programmingImprinting (organizational theory)Hemogenic endotheliumSex CharacteristicsBiological Variation Individualbiologybusiness.industryNeurodegenerationGene Expression Regulation DevelopmentalNeurodegenerative Diseasesmedicine.diseaseLife stage030104 developmental biologyHistonePrenatal stressbiology.proteinMicrogliabusinessNeuroscienceBiomarkers030217 neurology & neurosurgeryDevelopmental BiologyMechanisms of Ageing and Development
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Maternal eNOS deficiency determines a fatty liver phenotype of the offspring in a sex dependent manner

2016

ABSTRACT Maternal environmental factors can impact on the phenotype of the offspring via the induction of epigenetic adaptive mechanisms. The advanced fetal programming hypothesis proposes that maternal genetic variants may influence the offspring's phenotype indirectly via epigenetic modification, despite the absence of a primary genetic defect. To test this hypothesis, heterozygous female eNOS knockout mice and wild type mice were bred with male wild type mice. We then assessed the impact of maternal eNOS deficiency on the liver phenotype of wild type offspring. Birth weight of male wild type offspring born to female heterozygous eNOS knockout mice was reduced compared to offspring of wil…

0301 basic medicineMaleCancer Researchmedicine.medical_specialtyHeterozygoteNitric Oxide Synthase Type IIIOffspringBiology03 medical and health sciencesGenomic ImprintingMiceSex FactorsEnosInternal medicineFetal programmingmedicineAnimalsEpigeneticsMolecular BiologyGeneFatty liverWild typeHeterozygote advantageDNA Methylationmedicine.diseasebiology.organism_classificationFatty LiverMice Inbred C57BL030104 developmental biologyEndocrinologyPhenotypeKnockout mouseeNOSCarbohydrate MetabolismFemaleEpigeneticsInstitut für ErnährungswissenschaftmetabolismResearch Paper
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As Long as the Breath Lasts: In Utero Exposure to Ramadan and the Occurrence of Wheezing in Adulthood.

2017

While prenatal exposure to Ramadan has been shown to be negatively associated with general physical and mental health, studies on specific organs remain scarce. In this study, we explored whether Ramadan exposure during pregnancy affects the occurrence of wheezing, a main symptom of obstructive airway disease. Using data from the Indonesian Family Life Survey collected between 1997 and 2008 (waves 2-4), we compared wheezing occurrence among adult Muslims who had been in utero during Ramadan with that in adult Muslims who had not been in utero during Ramadan. Wheezing prevalence was higher among adult Muslims who had been in utero during Ramadan, independent of the pregnancy phase in which t…

AdultMalePediatricsmedicine.medical_specialtyAdolescentEpidemiologyIslam03 medical and health sciencesYoung Adult0302 clinical medicinePregnancy030225 pediatricsEpidemiologymedicinePrevalenceHumans030212 general & internal medicineLongitudinal StudiesFetal programmingPrenatal exposureRespiratory SoundsPregnancyFetusbusiness.industrySmokingFastingMaternal Nutritional Physiological PhenomenaMiddle Agedmedicine.diseaseMental healthFamily lifeIn uteroIndonesiaPrenatal Exposure Delayed EffectsFemalebusinessAmerican journal of epidemiology
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INVESTIGATION INTO THE MECHANISMS UNDERLYING THE TRANSGENERATIONAL EFFECTS OF MATERNAL HIGH-FAT DIET-INDUCED DYSBIOSIS ON OFFSPRING BRAIN AND METABOL…

2021

Genetic and environmental factors, and their interactions, contribute to the etiology and pathophysiology of neurodevelopmental disorders (NDDs), such as autism spectrum disorder (ASD). The clinical heterogeneity and phenotypic variability in patients with NDDs have made identification of causal mechanisms contributing to their onset difficult. Yet, unraveling the underlying causes of NDDs is essential to the development of appropriate preventive/therapeutic strategies. Maternal obesity is considered one of the main nongenetic risk factors for NDDs in progeny. We and others have found that high-fat (HF) obesogenic diets in both humans and animal models induce significant modifications in ma…

Gut microbiomeSocial determinants of healthSettore MED/07 - Microbiologia E Microbiologia ClinicaNeurodevelopmental disorderFetal programmingVertical transmissionPrenatal probioticsMaternal dietSettore MED/26 - NeurologiaSocial behavior
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Parental experience of a risky environment leads to improved offspring growth rate.

2014

Abstract Parasites (or diseases) are major selective force for the evolution of life history traits and parasite-host evolution. Mothers can show a variety of responses to parasites during pregnancy with different consequences for them or their offspring. However, whether information in the maternal environment before pregnancy can cause a change in the phenotype of the offspring is unknown. To avoid the confounding effect of pathogens and to reduce the risk of direct effect of mother's immune activation, we injected female laboratory mice with lipopolysaccharides (LPS) before mating. In order to provide a constant information on the potential infectious risk of the environment, females wer…

LipopolysaccharidesMale0106 biological sciencesPhysiologyOffspringPhysiologyAquatic ScienceBiology010603 evolutionary biology01 natural sciencesLife history theoryMice03 medical and health sciencesMaternal effectPregnancyFetal programmingmedicine[ SDV.EE.IEO ] Life Sciences [q-bio]/Ecology environment/SymbiosisAnimalsBody SizeSexual maturityMolecular BiologyThrifty gene hypothesisEcology Evolution Behavior and Systematics030304 developmental biologyInflammation0303 health sciencesPregnancy[ SDE.BE ] Environmental Sciences/Biodiversity and EcologyRodentReproductive successMaternal effectmedicine.diseasePaternal ExposureMaternal ExposureImmune SystemInsect SciencePaternal ExposureImmunologyFemaleAnimal Science and Zoology[SDE.BE]Environmental Sciences/Biodiversity and EcologyParent–offspring conflict[SDV.EE.IEO]Life Sciences [q-bio]/Ecology environment/Symbiosis
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Early life determinants of frailty in old age : the Helsinki Birth Cohort Stud

2018

Background there is evidence suggesting that several chronic diseases have their origins in utero and that development taking place during sensitive periods may affect the aging process. We investigated whether early life determinants would be associated with frailty in old age. Methods at a mean age of 71 years, 1,078 participants belonging to the Helsinki Birth Cohort Study were assessed for frailty according to the Fried frailty criteria. Early life measurements (birth weight, length, mother body mass index [BMI] and parity) were obtained from birth, child welfare and school health records. Multinomial regression analysis was used to assess the association between early life determinants…

MaleAgingSocial Determinants of HealthMaternal HealthBody Mass Indexolder people0302 clinical medicinePregnancyRisk FactorsPrevalenceMedicineEconomic Status030212 general & internal medicineFinland2. Zero hungerlife courseConfoundingAge Factorsta3141General Medicineta3142sikiö3. Good healthParityCohortLife course approachFemaleterveysBirth weightFrail ElderlyfrailtyRisk Assessment03 medical and health sciencesHumanssyntymäpainoOccupationsSocioeconomic statusAgedbusiness.industryInfant Newbornbirth weightikääntyminenfetal programmingSocial ClassAgeingageingRelative riskGeriatrics and GerontologybusinessBody mass index030217 neurology & neurosurgeryhauraus-raihnausoireyhtymäDemographyAge and Ageing
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Role of angiotensin II in arterial pressure and renal hemodynamics in rats with altered renal development: age- and sex-dependent differences

2012

Numerous studies have demonstrated that angiotensin II (ANG II) is involved in hypertension and renal changes occurring as a consequence of an adverse event during renal development. However, it was unknown whether this involvement is sex and age dependent. This study examines whether the increments in arterial pressure (AP) and in the renal sensitivity to ANG II are sex and age dependent in rats with altered renal development. It also evaluates whether the ANG II effects are accompanied by increments in AT1 receptors and oxidative stress. Experiments were performed in 3- to 4- and 10- to 11-mo-old rats treated with vehicle or an AT1 receptor antagonist (ARAnp) during the nephrogenic perio…

MaleAgingmedicine.medical_specialtyPhysiologyTetrazolesRenal functionKidneyAge and sexmedicine.disease_causeReceptor Angiotensin Type 1Rats Sprague-DawleyInternal medicinemedicineAnimalsArterial PressureRenal hemodynamicsFetal programmingAdverse effectSex CharacteristicsChemistryAngiotensin IIBiphenyl CompoundsAngiotensin IIMesenteric ArteriesRatsOxidative StressEndocrinologyBlood pressureBenzimidazolesFemaleAngiotensin II Type 1 Receptor BlockersOxidative stressAmerican Journal of Physiology-Renal Physiology
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Fetal growth restriction: A growth pattern with fetal, neonatal and long-term consequences

2019

Fetal growth restriction (FGR) or intrauterine growth restriction (IUGR) are the terms used for a fetus which has not attained its full growth potential for gestational age. FGR is a multifactorial syndrome responsible for increased fetal and neonatal morbidity and mortality as well as long term adverse outcomes involving auxological, metabolic, organic and functional domains. Clinicians distinguish early and late onset FGR, in relation to specific fetal anthropometric parameters related to the possible primary etiology and to different patterns of placental and maternal cardiovascular pathologies. Delivery of an early onset FGR or growth impaired newborn with congenital pathology should be…

Ponderal indexFetal growth restrictionFetal programmingTwinsTwinDevelopmental impairmentBrain sparing
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FETAL GROWTH RESTRICTION: A GROWTH PATTERN WITH FETAL, NEONATAL AND LONG-TERM CONSEQUENCES

2019

Fetal growth restriction (FGR) or intrauterine growth restriction (IUGR) are the terms used for a fetus which has not attained its full growth potential for gestational age. FGR is a multifactorial syndrome responsible for increased fetal and neonatal morbidity and mortality as well as long term adverse outcomes involving auxological, metabolic, organic and functional domains. Clinicians distinguish early and late onset FGR, in relation to specific fetal anthropometric parameters related to the possible primary etiology and to different patterns of placental and maternal cardiovascular pathologies. Delivery of an early onset FGR or growth impaired newborn with congenital pathology should be…

lcsh:R5-920fetal programmingFetal growth restrictionponderal indextwinsdevelopmental impairmentlcsh:Medicine (General)brain sparingEuromediterranean Biomedical Journal
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